ITFG1 Gene Summary [Human]

Located in extracellular exosome. [provided by Alliance of Genome Resources, Apr 2022]

Details

Type
Processed Transcript
Official Symbol
ITFG1
Official Name
integrin alpha FG-GAP repeat containing 1 [Source:HGNC Symbol;Acc:HGNC:30697]
Ensembl ID
ENSG00000129636
Bio databases IDs NCBI: 81533 Ensembl: ENSG00000129636
Aliases integrin alpha FG-GAP repeat containing 1, T cell immunomodulatory protein, LINKIN
Synonyms
2310047C21Rik,CDA08,D8Wsu49e,integrin alpha FG-GAP repeat containing 1,integrin α FG-GAP repeat containing 1,LINKIN,LNKN-1,LOC103694297,T-cell immunomodulatory,TIP
Species
Human, Homo sapiens

Protein Domains

A protein domain is a distinct structural or functional region within a protein that can evolve, function, and exist independently of the rest of the protein chain. These domains in human ITFG1 often fold into stable, three-dimensional structures and are associated with specific biological functions, such as binding to DNA, other proteins, or small molecules.
  • FG-GAP-like repeat

Top Findings

The most significant associations for this gene, including commonly observed domains, pathway involvement, and functional highlights based on current data.
disease
  • Huntington disease
  • rheumatoid arthritis
regulated by
  • megakaryocytes

Subcellular Expression

Locations within the cell where the protein is known or predicted to be active, providing insight into its function and cellular context.
  • Plasma Membrane
  • Extracellular Space
  • cellular membrane

Gene Ontology Annotations

Describes the biological processes, cellular components, and molecular functions associated with the human ITFG1 gene, providing context for its role in the cell.

Cellular Component

Where in the cell the gene product is active
  • extracellular vesicular exosome
  • plasma membrane

Gene-Specific Assays for Results You Can Trust

Streamline your workflow with assays designed for this gene. Our targeted dPCR and qPCR assays help you generate meaningful data – efficiently and accurately.